Skyscape Web Viewer : Stoelting's Handbook of Pharmacology and Physiology in Anesthetic Practice : Image Index

View All
Effect of Hydrostatic Pressure on Venous Pressures Throughout the Body
Effect-Site Concentrations With Traditional Versus Patient-Controlled Opioid Dosing
Effects of Human Growth Hormone Manifesting As Direct Effects or Via Production of Somatomedins in the Liver
Electrolyte Compositionchr(10)of Body Fluid Compartments
Etomidate, but Not Thiopental, is Associated With Decreases in the Plasma Concentrations of Cortisol
Excess Protamine Contributes to Elevations in the Activated Clotting Time (ACT), at Excesses of the Exact Dose Required to Reverse Systemic Anticoagulation Thus, Overdosage of Protamine Should Be Strictly Avoided
Fentanyl and Alfentanil Arterial Concentrations (Circles) and Electroencephalographic (EEG) Response (Irregular Line) to an Intravenous Infusion Alfentanil Shows a Less Time Lag Between the Rise and Fall of Arterial Concentration and the Rise and Fall of EEG Response Than Fentanyl Because It Equilibrates With the Brain More Quickly
Fetal-Maternal Arterial (Fa/Ma) Lidocaine Ratios are Greater During Acidemia Compared With a Normal pH
First-Degree Av Block
Gamma-Cyclodextrin (A) and Sugammadex (Modified gamma-Cyclodextrin) (B)
Gastric Emptying of Liquids is Exponential, Whereas Emptying of Solids is a Linear Process
Endogenous Corticosteroids
Ester and Amide Local Anesthetics
Events Triggered by Hypoxia Leading to Cell Death
Factors Affecting the Distribution of Pulmonary Blood Flow During One-Lung Ventilation (OLV)
Fentanyl Infusion Rate to Maintain a Plasma Concentration of 1 chr(956)G/Hr The Rate Starts Off Quite High Because Fentanyl is Avidly Taken Up by Body Fat
Fibrinogen is Converted to Fibrin that Polymerizes by the Action of Thrombin The Electron Micrograph Shows a Fibrin Clot With Red Blood Cells Trapped
Fraction of Halothane Removed During Passage through the Liver at Progressively Decreasing Alveolar Concentrations
Gas Exchange is Maximally Effective in Normal Lung Units With Optimal Ventilation to Perfusion (V/Q) Relationships
Glucagon Stimulates Tissue Release of Glucose, Free Fatty Acids, and Ketoacids and Hepatic Uptake of Amino Acids
Hematocrit Greatly Influences the Viscosity of Blood
Graphic Representation of the Typical Triphasic Core Temperature Pattern that Occurs after Induction of Anesthesia Note that the Phase 3 Plateau May Not Occur, Particularly During Regional Anesthesia or During Combined Regional and General Anesthesia
Hemodynamic Response to Rapid Intravenous Injection of Phenylephrine in a Single Patient
Hepatic Damage May Occur in the Rat Model after Administration of Inhaled or Injected Drugs When the Inhaled Oxygen Concentration is 10% Conversely, Hepatic Damage Occurs after Administration of Halothane, but Not Enflurane or Isoflurane, When the Inhaled Concentration of Oxygen is 12% or 14%
Hydration of Carbon Dioxide Results in Carbonic Acid (H2co3), Which Can Subsequently Dissociate into Bicarbonate and Hydrogen Ions
Impact of Surgical Stimulation on the Resting Paco2 (Mm Hg) During Administration of Isoflurane or Halothane
Individual and Mean (± Sd) Plasma Potassium (K-) Concentrations Determined 1 to 3 Days Preoperatively and Immediately Before the Induction (Preinduction) of Anesthesia
Inhalation of Nitric Oxide
Inhaled Anesthetics Introduced into Clinical Practice Beginning With the Successful Use of Nitrous Oxide in 1844 for Dental Anesthesia Followed by Recognition of the Anesthetic Properties of Ether in 1846 and of Chloroform in 1847 Modern Anesthetics, Beginning With Halothane, Differ from Prior Anesthetics in Being Fluorinated and Nonflammable Inhaled Anesthetics Introduced into Clinical Practice Beginning With the Successful Use of Nitrous Oxide in 1844 for Dental Anesthesia Followed by Recognition of the Anesthetic Properties of Ether in 1846 and of Chloroform in 1847 Inhaled Anesthetics
Initiation, Amplification, Propagation, and Stabilization of Hemostasis and Clot Formation

NDX