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The Simple View of Receptor Activation Also Explains the Action of Antagonist In This Case, the Antagonist (Red) Binds to the Receptor, but the Binding Does Not Cause Activation
The Sites of Action of the Different Diuretics
The Substitution of Nitrous Oxide for a Portion of Isoflurane Produces Less Decrease in Blood Pressure Than the Same Dose of Volatile Anesthetic Alone
The Synaptic Mechanism Underlying Peripheral, Nociceptive, Stimuli-Induced, and Persistent Heterosynaptic Potentiation of Dorsal Horn Neurons Transmitters and Mediators Released from Primary Afferents and Surrounding Microglial Cells, Including Substance P, Neurotrophins, and Cytokines May Act at a Distance on Dorsal Horn Neurons to Produce Long-Lasting Heterosynaptic Potentiation of Glutamatergic Transmission
The Synaptic Vesicle Exocytosis-Endocytosis Cycle
The Three Compartment Model from an Added Effect Site to Account for the Equilibration Delay Between the Plasma Concentration and the Observed Drug Effect The Effect Site Has a Negligible Volume
The Two Forms of Monoamine Oxidase Enzyme (MAO-A and MAO-B) Exhibit Substrate Selectivity
Third-Degree Atrioventricular Heart Block Occurring at an Infranodal Level (QRS Complexes are Wide)
Third-Degree Atrioventricular Heart Block Occurring at the Level of the Atrioventricular Node (QRS Complexes are Narrow)
Thromboelastography Recordings Obtained With the Rotem Device after the Addition of Rfviia and/or Fibrinogen in the Presence of Tissue-Type Plasminogen Activator in Volunteer Plasma Tissue-Type Plasminogen Activator Was Added to Stimulate Fibrinolysis
Transfusion-Related Acute Lung Injury (TRALI)
Transport Maximum for Glucose
Typical Time Course of Plasma Concentration Following Bolus Injection of an Intravenous Drug, With a Rapid Phase (Red), an Intermediate Phase (Blue), and a Slow Log-Linear Phase (Green) The Simulation Was Performed With the Pharmacokinetics of Fentanyl
Unipolar Limb Lead Circuit (VR)
Ventilatory Response Curves from One Individual
Ventricular Fibrillation
Ventricular Function Curves (Frank-Starling Curves) Depict the Volume of Forward Ventricular Ejection (Cardiac Output) at Different Atrial Filling Pressures and Varying Degrees of Myocardial Contractility
Ventricular Tachycardia
Visceral Innervation
Wave of Depolarization
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