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Yao & Artusio's Anesthesiology: Problem-Oriented Patient Management
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Triple-vessel coronary artery disease
D.4. What are the long-term outcomes of lung transplantation?
A.1. What is triple-vessel CAD? Name the branches of the coronary arteries.
Tropicamide (Mydriacyl)
Troponins
in subarachnoid hemorrhage
in perioperative myocardial infarction
See Traumatic subarachnoid hemorrhage
A.2. What are the associated cardiovascular defects in 22q11 deletion syndrome?
A.8. What is the pathophysiology of the IAA syndrome?
A.4. What is the pathophysiology of TOF with pulmonary atresia?
See Transthoracic echocardiography (TTE)
A.10. What is the differential diagnosis of d-TGA and how is the diagnosis made?
A.8. What are the different ways to estimate CO?
D.1. In the intensive care unit (ICU) 4 hours later, the patient became hypotensive with a low cardiac output. How could you distinguish between cardiac tamponade and pump failure? How would the TEE images differ?
A.1. What is thoracic aortic aneurysm (TAA)? How does it typically present? How is it diagnosed?
A.6. What are the basic principles of echocardiography? What are M-mode, B-mode, and Doppler color modalities? How are pressure gradients measured by echocardiography? How do TTE and transesophageal echocardiography (TEE) differ?
B.3. What are the preoperative considerations for the anesthesiologist before TAA repair?
E.3. How is transcatheter AV replacement performed?
Valvular Heart Disease
Patent Ductus Arteriosus and Prematurity
Transmission spectrometry
Traumatic brain injury
Traumatic wet lung
Triceps skinfold thickness test
Trigger point injection, for low back pain
Trismus
Truncus arteriosus, in 22q11 deletion syndrome
Tuberous sclerosis
Tubocurarine
C.13. How are neuromuscular blocking agents, such as succinylcholine, atracurium, cisatracurium, vecuronium, pancuronium, rocuronium, and curare, metabolized and eliminated? Which have significant histamine release?
C.12. How does burn injury affect the dosing of nondepolarizing neuromuscular blocking agents?
Turcot syndrome
TURP
See Tranexamic acid (TXA)
C.10. You decide to use rotational thromboelastometry (ROTEM) to guide your resuscitation efforts. After 20 minutes, the results of the ROTEM are normal; however, the field is still bloody. The surgeon suggests tranexamic acid (TXA) for the patient. What is TXA and what is its mechanism of action? Is TXA effective at reducing blood loss in major spine surgery? What are the risks of TXA administration? Is TXA an appropriate choice in this patient?
C.21. What concentrated factors and hemostatic agents are available for bleeding patients?
C.9. Two hours later, estimated blood loss (EBL) is approximately 2 L and the patient has been resuscitated with 3 units of packed red blood cells (pRBCs), 229 mL intraoperative cell salvage (cell saver), and 500 mL albumin in addition to 4 L crystalloid. The surgeon says the field is still "oozy" and there is ongoing blood loss. What are your concerns for resuscitation moving forward? How will you guide your resuscitation?
C.2. Discuss the indications and use of available antifibrinolytic drugs. What dose should be given? Describe the side effects of antifibrinolytic therapy.
C.7. When should this patient receive tranexamic acid (TXA)?
C.I-17. What antifibrinolytic will you use to reduce blood transfusions?
Placenta Previa/Placenta Accreta Spectrum
Major and Complex Spine Surgery
See Tranexamic acid (TXA)
T-tube test
Tumor embolization
Turner, aortic aneurysms with
Two-wavelength pulse oximetry
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